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The impact of risky anesthetics on respiratory system resistance in patients with persistent obstructive pulmonary disease. Wedel DJ, Gammel SA, Milde JH, Laizzo PA. Delayed onset of malignant hyperthermia induced by isoflurane and desflurane in contrast with halothane in susceptible swine. Tuchy GL, Tuchy E, Bleyberg M, Smith GL, Calava J. Pharmacodynamics of mivacurium throughout desflurane anesthesia. Kumar N, Mirakhur RK, Symington MJ, Loan PB, Connolly FM. A comparability of the results of isoflurane and desflurane on the neuromuscular effects of mivacurium. Strebel S, Lam AM, Matta B, Mayberg TS, Aaslid R, Newell DW. Dynamic and static cerebral autoregulation during isoflurane, desflurane, and propofol anesthesia. Karzai W, Haberstroh J, Priebe HJ. The effects of increasing concentrations of desflurane on systemic oxygenation throughout one-lung air flow in pigs.
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Meta-analysis of average and variability of time to extubation evaluating isoflurane with desflurane or isoflurane with sevoflurane. Heavner JE, Kayel AD, Lin BK, King T. Recovery of aged patients from two or more hours of desflurane or sevoflurane anaesthesia. Martin JL, Plevak DJ, Flannery KD, Charlton M, Poterucha JJ, Humphreys CE, et al. Kawaguchi M, Sakamoto T, Ohnishi H, Shimizu K, Karasawa J, Furuya H. Intraoperative myogenic motor evoked potentials induced by direct electrical stimulation of the exposed motor cortex under isoflurane and sevoflurane. Matta BF, Mayberg TS, Lam AM. Direct cerebrovasodilatory results of halothane, isoflurane, and desflurane during propofol-induced isoelectric electroencephalogram in humans.
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During the upkeep phase of anesthesia, desflurane provides exact management and speedy titrations within the anesthetic depth and rising concentrations of desflurane produce dose-dependent decreases in BP. Small concentrations (5–10% of MAC) can impair pharyngeal function and the ability to handle international material in the pharynx. McKay et al. demonstrated that the extra rapid and predictable restoration of desflurane in contrast with sevoflurane also applies to the return of protective airway reflexes in overweight/obese sufferers . The respiratory system may be adversely affected by inhalational anesthetic brokers by inflicting respiratory melancholy, airway irritation, and bronchospasm. In healthy volunteers, at concentrations up to 1.66 MAC, desflurane produced a dose-dependent decrease in tidal volume with a rise in respiratory frequency.
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Absence of hepatotoxicity is consistent with the minimal biodegradation of desflurane, the sustained hepatic arterial blood circulate, and the speedy elimination of desflurane after termination of anesthesia. A current study in rats demonstrated that unlike halothane, biodegradation of desflurane produces no covalently-bound fluorine and thus the potential for hepatotoxicity seems to be low. This low potential has additionally been shown in human volunteers and sufferers with postanesthetic renal operate tests in human volunteers and patients showing no change after desflurane anesthesia. In the realm of educating, Dr. Heavner presently serves on the school on the University of South Alabama as a professor of saxophone, music training and jazz research. He is a full member of the graduate college and space coordinator for all music schooling programs in the department of music.
